Bruce, D, Fu, SL, Armstrong, S & Dean, RT 1999, 'Human apo-lipoprotein B from normal plasma contains oxidised peptides', INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY, vol. 31, no. 12, pp. 1409-1420.
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Davies, MJ, Fu, SL, Wang, HJ & Dean, RT 1999, 'Stable markers of oxidant damage to proteins and their application in the study of human disease', FREE RADICAL BIOLOGY AND MEDICINE, vol. 27, no. 11-12, pp. 1151-1163.
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McNevin, D, Barford, J & Hage, J 1999, 'Adsorption and biological degradation of ammonium and sulfide on peat', Water Research, vol. 33, no. 6, pp. 1449-1459.
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Petterd, CI, Hamshere, J, Stewart, S, Brinch, K, Masi, T & Roux, C 1999, 'Glass particles in the clothing of members of the public in south-eastern Australia – a survey', Forensic Science International, vol. 103, no. 3, pp. 193-198.
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This study was undertaken to test the validity of the proposal that there is a natural background level of glass particles on the surface of clothing of members of the community. A total of 2008 upper outer garments collected from random members of the p
Roux, C, Gill, K, Sutton, J & Lennard, C 1999, 'A further study to investigate the effect of fingerprint enhancement techniques on the DNA analysis of bloodstains', Journal of Forensic Identification, vol. 49, no. 4, pp. 357-376.
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This study investigated the effect of common and well established fingerprint enhancement techniques on the subsequent DNA analysis of items potentially bearing both fingerprints and biological evidence. Bloodstains of varying ages were prepared on different surfaces and various fingerprint enhancement techniques were applied to the samples. DNA typing was performed using PCR amplification (D1S80 and CTT system). The results showed that magnetic powder, multimetal deposition (MMD) and ultraviolet (UV) radiation are not recommended for use in a sequence of analyses involving DNA typing. Strong white light, white and aluminum fingerprint powders, physical developer (PD) after 1,8-diaza-9-fluorenone (DFO), PD after ninhydrin with cadmium (Cd) salt treatment, and cyanoacrylate with gentian violet or Ardrox stains may be used successfully in a sequence of analyses involving DNA typing. Ninhydrin with secondary metal salt treatment, DFO, amido black, diaminobenzidine (DAB), black powder, Stickyside Powder, cyanoacrylate with rhodamine stain, and luminol may be used before DNA analysis but care must be taken to ensure that sufficient DNA is extracted and analyzed.
Roux, C, Novotny, M, Evans, I & Lennard, C 1999, 'A study to investigate the evidential value of blue and black ballpoint pen inks in Australia', Forensic Science International, vol. 101, no. 3, pp. 167-176.
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The aim of this project was to investigate the evidential value of blue and black ballpoint pen inks in Australia. For this purpose, 49 blue and 42 black ballpoint pen inks, of different brands, models and batches, representative of those ballpoint pens
Sanni, LA, Fu, SL, Dean, RT, Bloomfield, G, Stocker, R, Chaudhri, G, Dinauer, MC & Hunt, NH 1999, 'Are reactive oxygen species involved in the pathogenesis of murine cerebral malaria?', JOURNAL OF INFECTIOUS DISEASES, vol. 179, no. 1, pp. 217-222.
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To investigate the involvement of oxidative tissue damage in the pathogenesis of murine cerebral malaria (CM), brain levels of protein carbonyls, 3,4-dihydroxyphenylalanine(DOPA), 0-tyrosine, and dityrosine were measured during Plasmodium berghei ANKA (PbA) and P. berghei K173 (PbK) infections. During PbA infection in a CM model, brain levels of the substances were similar to those in uninfected mice. The role of phagocyte-derived reactive oxygen species in the pathogenesis of CM was examined in $gp91_{phox}$ gene knockout mice. The course of CM in these mice was the same as in their wild type counterparts. To examine whether superoxide production in the central nervous system could have occurred via increased xanthine oxidase activity, brain concentrations of urate were measured in CM mice and in mice infected with PbK (which does not cause CM). Brain urate concentration increased significantly in both groups of mice, suggesting that purine breakdown is not specific to CM. These results indicate that reactive oxygen species probably do not contribute to the pathogenesis of murine CM
